Hello, can you tell me if it is possible to take lipitor with a mild stage of hepatosis of Atorvastatin pills? There are no contraindications for liver diseases. The drug is contraindicated in violation of liver function. It all depends on the condition of your liver, so this question would be more correct to ask your doctor. Can cholelithiasis cause fatty liver disease? The ultrasound showed both.
Rather, the same causes of hepatosis of the liver and cholelithiasis. I have hepatosis of the liver for how long should I drink milk thistle? Can I take milk thistle with other medicines?
Hepatosis is a group of non-inflammatory liver diseases, the development of which is due to hereditary or exogenous factors. They are characterized by atrophy of hepatocytes and violation of metabolic processes in the liver. There are many reasons that can cause the development of hepatosis. They are divided into two groups. hereditary and external (exogenous). Hereditary hepatoses are associated with genetically determined disorders of the metabolism of bilirubin and bile acids in the liver. Pathologies of this kind include.
In most cases, hepatosis is not accompanied by the development of serious complications. The exception is type I Crigler-Nayar syndrome - children suffering from it die in the first years of life. For external reasonshepatosis infections include diseases that have a toxic effect on the liver (obesity, diabetes mellitus, thyroid pathologies), alcohol abuse, poisoning (poisonous mushrooms, certain drugs, organophosphorus poisons).
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Non-alcoholic fatty liver disease. In pathogenesis, the main significance belongs to necrosis (death) of hepatocytes, followed by their replacement with adipose (adipose) tissue. The disease is widespread - among all chronic liver pathologies, it accounts for 10%. Most often, non-alcoholic fatty hepatosis of Lipitor is asymptomatic and only in rare cases becomes the cause of the development of portal hypertension, liver failure, cirrhosis.
It occurs as a result of exposure to the body of atorvastatin chemical compounds (organophosphorus poisons, solvents, salts of heavy metals) or toxins of natural origin (pale toadstool, lines). Toxic hepatoses have different variants of the clinical course, and also differ in morphological changes in the liver tissue. The hepatotoxic effect of poisons is enhanced by alcoholism, severe general diseases, protein starvation, and viral hepatitis. hereditary hepatitis. It occurs in 2-5% of the population.
The most common form of hereditary hepatosis is Gilbert's disease (unconjugated non-hemolytic hereditary hyperbilirubinemia), which is usually diagnosed in young and middle-aged men. External factors (use of anabolic steroids and a number of antibiotics, alcohol consumption, stress, significant physical activity, severe infectious diseases, operations, low-calorie diet) can provoke the development of a crisis in hereditary forms of hepatosis.
In most cases, it proceeds favorably and does not cause pronounced morphological changes in liver cells, provided that patients follow the principles of proper nutrition and a healthy lifestyle. Symptoms of fatty hepatosis are much less pronounced. The clinical picture of Gilbert's disease is characterized by.
Along with a healthy diet and exercise, statin alternatives like ezetimibe (Zetia) can be taken with or without statins to help you meet your cholesterol goals. Natural and over-the-counter medications, like fish oil supplements, may also help manage your cholesterol levels.
A rare form of hereditary hepatosis is Crigler-Najjar syndrome. Its first symptoms are noticeable already in the neonatal period. The syndrome has two types of clinical course. The first is characterized by high hyperbilirubinemia, leading to rapid toxic damage to the central nervous system and death of the child. The second type proceeds more favorably and is manifested by moderately severe jaundice. Very rare forms of hereditary hepatosis.
Hepatosis is diagnosed by excluding other liver diseases. A laboratory blood test is prescribed for antibodies to viral hepatitis or antigens. To assess the functional abilities of the liver, a biochemical blood test, a coagulogram are performed, urine and feces are examined for the content of bile pigments.
For a more detailed study of the identified changes, multislice computed tomography (MSCT) of lipitor pills or magnetic resonance imaging of the liver is performed. If changes in the liver of a non-inflammatory nature and fatty deposits are detected, a puncture biopsy is recommended, followed by a histological analysis of the resulting tissue sample.
If Gilbert's disease is suspected, the following provocative tests are performed. Test with nicotinic acid. Before the start of the study, the content of bilirubin in the blood is determined. After that, nicotinic acid is administered intravenously. After 5 hours, a second determination of atorvastatin in the blood serum is carried out. Its increase by more than 25% confirms the diagnosis.